Wednesday, June 12, 2013

What Is Anthony Kennedy Thinking?

Why the Supreme Court justice might decide we’ve been thinking about gay marriage all wrong.

Supreme Court watchers have long made a national sport out of parsing Justice Anthony Kennedy’s every word. From issues as diverse as the death penalty, terrorism, and gay rights, Kennedy has been the only conservative justice to vote with the court’s more liberal wing. It’s not surprising, therefore, that as we wait for the court’s decision on same-sex marriage bans, the search for clues to Kennedy’s thinking has shifted into high gear.
What is surprising, however, is that in this quest for insights into Kennedy’s frame of mind, pundits have virtually ignored one of the few things he flat-out told us about his views on same-sex marriage.
In March, during the oral argument about California’s same-sex marriage ban, Kennedy said that he was “trying to wrestle” with a “difficult question” about the constitutionality of same-sex marriage. The question on his mind was whether prohibitions on same-sex marriage are a form of gender discrimination. The lawyer defending the ban, Charles Cooper, responded that this was a case about sexual orientation, not gender, and the argument quickly moved in a different direction.
But we shouldn’t dismiss Kennedy’s question about gender discrimination too hastily. The court’s precedents on gender might offer Kennedy the conservative compromise he is looking for: a way to recognize a constitutional right for same-sex marriage in a limited way.
The gender-discrimination argument is not complicated. Imagine Alice applies for a license to marry Charlie and it is granted. Yet if Bob applied for a license to marry Charlie, he would be denied. The crucial difference between Alice and Bob is, of course, their gender—not their sexual orientation. In fact, as we all know, homosexuals have long been free to marry members of the opposite sex. Thus, Kennedy is wrestling with the possibility that Bob is being discriminated against because he is a man and not because he is gay. And, if so, should the court apply the same level of heightened protection it traditionally applies whenever the government treats men and women differently?
Kennedy wouldn’t be the first to see the denial of marriage licenses to same-sex couples as gender discrimination. A plurality of the Supreme Court of Hawaii accepted this argument in a 1993 case and held that the state’s ban on same-sex marriage violated the state’s constitution (although a constitutional amendment allowing marriage to be limited to opposite-sex couples was later upheld.) Judge Stephen Reinhardt of the 9th Circuit Court of Appeals similarly held in a 2009 order that denying a federal employee the ability to name his husband as his beneficiary amounted to sex-discrimination because the designation would have been allowed had he been a woman. Law professor Andrew Koppleman made the same argument in a New York University law review article, explaining, “[a]s a matter of definition, if the same conduct is prohibited or stigmatized when engaged in by a person of one sex, while it is tolerated when engaged in by a person of the other sex, then the party imposing the prohibition or stigma is discriminating on the basis of sex.”
A standard response to the gender-discrimination argument is that it’s not discrimination if both genders are denied the same benefit. In other words, because all men can only marry women and all women can only marry men, everyone is being treated equally. But this response is easily rebutted.
Laws that once prohibited interracial marriage were often described in the same “everybody loses” terms. Take, for example, Virginia’s anti-miscegenation law, which the Supreme Court declared unconstitutional in 1967 in Loving v. Virginia. The Virginia law declared it a crime if “any white person intermarry with a colored person, or any colored person intermarry with a white person.” Virginia argued to the Court that because the law punished “equally both the white and the Negro participants in an interracial marriage,” there was no Equal Protection violation. White people were free to get married, just not to nonwhites and vice versa. The court disagreed, and declared the law to be racially discriminatory.
During the arguments over California’s Proposition 8 banning gay marriage, Cooper’s response to Kennedy’s question offered another common, yet flawed, retort to the gender-discrimination argument. Cooper said that this case involves a gender-based classification only “in the sense that marriage itself is a gendered institution, a gendered term.” The government in the Hawaii case similarly argued “the right of persons of the same sex to marry one another does not exist because marriage, by definition and usage, means a special relationship between a man and a woman.”
Surely Kennedy could easily see through this kind of circular logic. Marriage is a “gendered term” that “by definition and usage” involves only members of the opposite sex precisely because we have always prohibited same-sex couples from marrying. In Loving, there was a similar reliance on the so-called natural state of marriage. The trial court judge declared “there was no cause for” interracial marriage because “God created the races … and did not intend for the races to mix.” But it proves nothing to say that marriage is innately one way and must remain that way when—whether because of alleged divine order or legal fiat—it has never had the opportunity to be any other way. Marriage is no more an inherently gendered institution than an inherently racial one.
The gender-discrimination framework may appeal to Kennedy in other ways, too. During oral argument, he expressed worry about the court about moving too far too fast. Bouncing between metaphors of entering “uncharted waters” or going off a “cliff” with its decision, Kennedy expressed a desire for the court to proceed cautiously “in light of the newness” of the issue.
This approach could help Kennedy with these concerns. He doesn’t have to break new legal ground by declaring a constitutional right to be free from discrimination based on sexual orientation. Instead, Kennedy could turn to the much more developed path of our constitutional protections against gender discrimination. The outcome (constitutional protection for same-sex marriages nationwide) would be revolutionary, but the basis for it (gender discrimination) would be familiar.
The reach of these cases is also naturally circumscribed. A gender-discrimination ruling on marriage would not, for example, determine how much constitutional protection a person might receive if he was fired from his job because of his sexual orientation. Kennedy could save that case for another day. It also does not give fodder to the slippery-slope argument about polygamy, which presents a problem of numbers and not gender.
Another advantage, at least perhaps in Kennedy’s worldview, is that his opinion need not hinge on a constitutional right to privacy. Kennedy could side-step any icky feelings he might get from wading into privacy rights, which tend to include family-based freedoms like the right of procreation, childrearing, contraception, and abortion. Instead he could rest easy that a gender-discrimination decision would put this case squarely in the Equal Protection chapter of future constitutional law textbooks.
Of course, we will know soon enough if Kennedy is really writing the court’s opinion on gay marriage or not. But if he does, and if he chooses to rely on the traditional framework of gender discrimination, we can’t say he didn’t try to warn us.

Text formatting on Google+ posts


https://www.google.com/search?sclient=psy-ab&q=google+plus+formatting+text&oq=google+plus+formatting&gs_l=serp.1.8.0l10.126.3942.3.8836.17.12.0.5.5.0.214.2223.0j11j1.12.0...0.0...1c.1.12.serp.MYHyZLsVIpc&pbx=1&biw=1083&bih=854&cad=cbv&sei=EOmLUaLBJuqU0QG7voCgBA

While we are waiting for a simple WYSIWYG -editor or similar to appear on Google+ (I'm sure it's being worked on) here are some shortcuts that you can use to format the post texts.

This is a great resource by Simon Laustsen:  In many different languages, too!
Malay, Chinese, Croatian, Finnish, Persian, Dutch, Italian, Swedish...

If you think your language deserves one, why not translate it?  Contact Simon at +Simon Laustsen

Sometimes you have to leave one or two extra spaces before the _ or - especially after a +mention or they might not work.  With paragraphs the characters have to be repeated, for instance when using  _italics_.  If this doesn't work, edit it again, perhaps deleting the characters including the spaces before and after the word / paragraph, then try again.

EDIT:
I've noticed that 3 spaces should be enough after the last word, but sometimes it works with only 2, especially when you are copy pasting and type the _ first.
I just commented on a post and wrote _blushes..._ with 2 spaces: It didn't work so I edited and added the third space and that worked.




More tips on creating readable posts


When you write a post, pay attention to the paragraphs.  I mean, use them.  Especially with longer posts it's very tiring to read a text that is all in one piece.


+Ahmed Zeeshan wrote a very nice piece about the structure and formatting of your longer posts:

» A blogging platform
One of the appealing features of google+ is the ability to write lengthy posts. Combine that with the excellent commenting system and we get ourselves a very functional blogging platform. However, there is also another mis-used feature which all g+ content curators stand to benefit from if used properly: Rich text formatting...

Often times I see very long posts that are a giant block of text with no formatting or structure applied to them. They might contain top quality content but because they're not presented in an appealing way, mosts users do not bother to read through them. The lack of engagement then surprises the original author. This is even true for some of our power users.

Google+ offers us the chance to get rid of the ridiculous TL;DR (too long; didn't read) culture on the internet. That will only happen if we make our posts easier and simpler to read. Our readers should be able to take one look at the post and see exactly what it is about. The structure of the post should draw them in and encourage them to read all of it.
Read more about the structure in his Google+ post .




Note: 


Now the web based +1 button (this one is for Chrome) also lets you add posts to your profile's +1 tab which has changed since the publishing of the Cheat Sheet.  With Pages it only adds the link to the Google+ page, but with individuals' posts you may open them from the time stamp in your browser and click on the external +1 button. The post is saved on your tab.

Note 2:  After the last Google+ Update in FEB13, the external Chrome +1 button seems not to save posts to +1 Tab on the profile page, again...  Hmm. Let's hope for a change.

More links and advice:

Think Google+ is a Ghost Town? You're using it Wrong, probably

+The G+ Resource  blogs about creating readable Google+ posts

What happens when you move your blog to Google+  by +Jay Acunzo

Quick video tip on formatting

Copypaste symbols if you want to decorate the text

Remember when posting: People love images.


When you are sharing links:  I'd advice you to use larger images added to the post instead of the links' own small ones in the linkbox, for attracting more attention if needed.

You may use a link shortener like http://goo.gl/ or http://bit.ly and add the link inside the text after you have uploaded or added (shared) the image from your Google+ albums.

Image is everything! :-)
Never just dump links without a headline and some snippet of the content or your own views and thoughts. Use hashtags related to your content, or create one unique for your own use.

Try to use a relevant picture and not do what I did here:  This is called "Linkbait" and has nothing to do with the matter in hand.  :-)


eb Applications Stack Exchange is a question and answer site for power users of web applications. It's 100% free, no registration required.
What is the markup language for creating posts (and responding to posts)?
I know of the following ones:
  • To make text bold, use stars around the text: *bold text*
  • To make text italics, use underscores: _italic text_
What else is there? Is there any way to insert bullet-lists? What about larger font sizes, etc?
Google+ is starting to become a blog of sorts, and having a fully featured markup language makes it very helpful for formulating useful and lengthy posts.
If no other native markups exist, is there any way to insert HTML? Ultimately, what are the various freedoms we have in making and replying to posts to markup the text?
shareimprove this question
1
so how do I type these chars like in code, and AVOID G+ turning them into stupid font highlights? massive fail if I can't put code in a G+ post... – Sam Watkins Feb 14 at 2:54


Unfortunately the markup for Google+ (and Google Talk by extension) is fairly limited.
You're missing:
  • -item- for strikethrough
  • @name or +name to tag someone.
See screenshot for more posting tips:
enter image description here
Reference: Posting Tips, section "Posting tips"
shareimprove this answer
1
The original Markdown doesn't have strikethrough I placed the markup you need for strikethrough above. – phwd Jan 23 '12 at 6:22
1
Are there any others? If this is all, Google+ could use some serious room to grow. At a minimum, I'd like to be able to do some simple formatting like Bullet and Number lists. – Jack Jan 23 '12 at 7:20
1
Nope, that's it. Bold, italic, and strikethrough. support.google.com/plus/bin/… – Al Everett Jan 24 '12 at 2:37
Ohh, if Google+ or any other social networking starts accepting formatted text, then it would all look like a mess, not comments. – Deepak Kamat Feb 5 at 12:03

Fibromyalgia Infomercial

Idiopathic disease is defined as one that develops without any apparent or known causes. That is the term used for fibromyalgia, autoimmune diseases, including lupus and chronic fatigue syndrome. While many of these diseases have recognizable signs and symptoms, the lack of causality haunts medical schools, doctors, practices and hospitals. The only ones benefiting from lifelong symptom treatments associated with chronic fatigue syndrome, lupus, autoimmune disease, or fibromyalgia are the pharmaceutical companies who sell billions in medication to treat them. A long list of pain medications, sleep-aids, anti-depressants, and anti-inflammatories are not sufficient because the diagnosis is incorrect. Instead, let's look at what the possible causes are to these diseases.

A Conventional Scientific Overview of CFS (Chronic Fatigue Syndrome) and Fibromyalgia Causatives

Below, is a quick list of idiopathic disease causes — we will give a clinical review and explanation as to what takes place.
·         Brain abnormalities
·         Genetic factors (HPA) axis
·         A hyper-reactive immune system
·         Viral or other infectious agents like (Chronic Lyme disease Complex)
·         Psychiatric or emotional conditions

Are Genetics to Blame?

Chronic Fatigue Syndrome and fibromyalgia have been linked with genes involved in the hypothalamic-pituitary-adrenal axis and the sympathetic nervous system. These genes regulate response to trauma, injury, and other stressful events. Our ten years of clinical experience shows that while such traumas could play a role in the etiology (the trigger to exhibiting symptoms) of the disease, they are unlikely the conditions’ causes.

What is the Hypothalamic-Pituitary-Adrenal Axis (HPA)? Does Lyme Disease Play a Role?

HPA makes up a multi-set of direct influences and feedback interactions among the hypothalamus, the pituitary gland (a pea-shaped structure located below the hypothalamus), and the adrenal, also called "suprarenal," glands which are small, conical organs on top of the kidneys.

The interactions among these organs constitute the HPA-axis, a major part of the neuroendocrine system. From here, the body regulates reactions to stress, as well as processes such as digestion, the immune system, mood, emotions, sexuality, as well as energy. Infectious disease, such as chronic Lyme disease complex, impacts the HPA-axis via neurotoxins that compete for the same receptor sites used by the HPA-axis.

In fact, such infections can bring about identical symptoms of some idiopathic diseases listed above and many of the symptoms associated therewith. This should bring our attention to chronic Lyme Disease complex, which is composed of a number of infections and neurotoxins that bring about even more symptoms than those listed earlier in this article.

Does HPA Affect Fibromyalgia and Chronic Fatigue Syndrome?

Abnormal levels of certain chemicals regulated in the HPA axis area of the brain system have been proposed as a cause of Chronic Fatigue Syndrome and also have some influence in fibromyalgia. This system controls important functions, including sleep, stress response, and depression. Of particular interest to researchers are the chemicals and other factors listed below that are controlled by the HPA-axis.

The HPA-axis is involved in the neurobiology of mood disorders and functional illnesses, including anxiety disorder, bipolar disorder, insomnia, post-traumatic stress disorder, borderline personality disorder, ADHD, major depressive disorder, burnout, chronic fatigue syndrome, fibromyalgia, irritable bowel syndrome and alcoholism. Antidepressants, which are routinely prescribed for many of these illnesses, serve to regulate HPA-axis function. All of these conditions and their symptoms are commonly seen in chronic Lyme disease patients that contain a host of infections and neurotoxins that block serotonin receptors in the brain.

Can Chronic Lyme Disease Complex or Infectious Disease Affect HPA?

Patients may have contracted an infection at any point in their lifetime. However, the symptoms of chronic Lyme disease complex or its coinfections may remain unseen or dormant until the individual is weakened by a trauma or trigger. This could be anything from childbirth or a car accident to the death of a loved one, a divorce or even a vaccine, as seen among children with weakened immune systems.

In the etiology of chronic infectious disease, the traumatic event is a trigger, but not the cause of autoimmune disease, Chronic Fatigue Syndrome, or fibromyalgia. Nevertheless, treating these triggers is critically important to the new patient's care. What we find is that infection and not genetic defects are at the root of HPA-axis disruption in the brain itself.

The Major Impact of Epigenetic Changes

A number of studies have found that alterations in genes are caused by infections involving immune function, intracellular communication and energy transfer. Researchers have identified many different genes in patients with Chronic Fatigue Syndrome that relate to blood disease, immune system function, and infection.

However, despite these identifications, there is no clear pattern to them and it is quite possible that it is the infections alone that are altering these genes and are responsible for impacting mental and emotional health as well. It is very possible that the infections can alter these genes that impact mental and emotional health as well.

Important Neurotransmitters Changed By Neurotoxins Competing For Receptor Sites

Some patients with Chronic Fatigue Syndrome have abnormally high levels of serotonin, a neurotransmitter (chemical messenger in the brain), and also show deficiencies in dopamine, an important neurotransmitter associated with feelings of reward. In some cases there is also a demonstrable imbalance between norepinephrine and dopamine.

A number of studies on Chronic Fatigue Syndrome have shown patients have lower cortisol levels, a stress hormone produced by the adrenal glands. It has been suggested that such cortisol deficiencies are responsible for Chronic Fatigue Syndrome patients having impaired or weakened responses to psychological or physical stresses like worry, infection, or exercise. However, administering replacement cortisol improves symptoms only in some patients. Why? Infection and their toxins (neurotoxins) must be cleared before hormone replacement can begin to be effective in these patients. It is also common for these patients to have thyroid, testosterone and cortisol issues.

Idiopathic Diseases at Root of Myriad Psychological Disorders Including Sleep-Related Ailments

Evidence suggests that certain CFS, fibromyalgia, and autoimmune patients have disturbed circadian rhythms (disorder of the sleep-wake cycle), which is regulated by the so-called circadian clock, a nerve cluster in the HPA-axis. These are commonly seen in chronic Lyme disease complex along with a number of other neurological symptoms.

A mentally or physically stressful event, such as a viral infection, may disrupt natural circadian rhythms. An inability to reset these rhythms results in a perpetual cycle of sleep disturbances. Medications that improve sleep can be very helpful for certain patients with Chronic Fatigue Syndrome, fibromyalgia and autoimmune diseases. But, until the infections are cleared and hormones are rebalanced, long-term improvement is unlikely, as the patient is likely to relapse.

Psychological, personality and social factors are strongly associated with Chronic Fatigue Syndrome, fibromyalgia, and autoimmune disease like lupus. There is a distinct complex relationship between physical and emotional factors.

What Specific Infections are Responsible

Because most of the features of Chronic Fatigue Syndrome resemble those of a lingering viral illness, many researchers have focused on the possibility that a virus or some other infectious agent, in some cases, causes the syndrome.

We have clinically determined that these patients usually have a group of viral, bacterial, parasitic and fungal infections that make up what we call Lyme Disease Complex. Some patients may or may not have actual Lyme disease but may have another type of tick-borne illness along with a host of co-infections that have brought about immunological, hormonal, and neuroendocrine changes.

Still, not all Chronic Fatigue Syndrome patients show signs of infection. And although experts have long been divided on whether infections play any role in this disorder at all, it does seem clear that subtypes of both viral and non-viral Chronic Fatigue Syndrome exist. That being said, researchers have seemingly overlooked the complexity of mute-infections, multi-toxins and heavy metal components that complicate these conditions, making them extremely difficult to diagnose on a case-to-case basis. When a complex of infections exists, they can affect the activation and replication of each other via biofilm communities. To be certain, most patients are never tested thoroughly and correctly for all the infections that make up chronic Lyme disease complex.

Infections Looking Like the Cause

The theory for Chronic Fatigue Syndrome having a viral cause is not based on hard evidence, rather, on an ever-growing series of observations. Chronic Fatigue Syndrome, as well as Fibromyalgia and Autoimmune disease patients, are often found with elevated levels of antibodies to many organisms that cause fatigue and other Chronic Fatigue Syndrome symptoms. Such organisms include those that cause Lyme disease, Candida ("yeast infection"), herpes virus type 6 (HHV-6), human T cell lymph tropic virus (HTLV), Epstein-Barr, measles, coxsackie B, cytomegalovirus, or parvovirus.

Many of these infectious agents are very common; however, none have emerged as a definitive cause of CFS. Well-designed studies of patients who met strict criteria for CFS without any known cause have not found an increased incidence of any specific infection(s).

In up to 80% of cases, CFS starts suddenly with a flu-like condition. In the U.S., there have been reports of cluster outbreaks of CFS occurring within the same household, workplace, and community (but most have not been confirmed by the Centers for Disease Control and Prevention). However, most cases of CFS occur sporadically in individuals and do not appear to be contagious. These all have the pattern of infections and more importantly, complexes of infections taking over the patient's immune system, which is clearly seen in the depressed CD57 markers found in almost all of this population.

Infection Complexes Leading to Immune System Abnormalities and Immuno-Compromised States

CFS is sometimes referred to as "Chronic Fatigue Immune Dysfunction Syndrome." In many cases, studies have detected many immune system irregularities. Some components appear to be over-reactive, while others appear to be under-reactive, but no consistent picture has emerged to explain CFS as a disease of the immune system in conventional medical practices. Chronic Lyme disease patients almost always have depressed CD57 marker called the striker panel and this is almost never run on chronic fatigue patients when they go to their doctor. Almost 100% of the time we find decreased key immune function in all CFS patients because we are running the correct diagnostics.

Autoimmunity Overlaps with Other Conditions

The risk profile for CFS is similar to the risk profiles for a number of autoimmune diseases. Studies are inconsistent with regards to the presence of auto-antibodies (antibodies that attack the body's own tissues) in CFS, so the disease is unlikely to be due to auto-immunity, making it more likely connected to infectious disease. In Lyme disease patients, we typically see that the patient was diagnosed at one time or another with several autoimmune diseases, but almost certainly the previous physicians were confused.